Causes of hemiplegic cerebral palsy and its management
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A retrospective population-based study of 169 instances with hemiplegic cerebral palsy (CP) from the South-western Swedish health care region covering the birth years 1969-1978 was conducted. The goal was to look at the prevalence, aetiology, and neurodevelopmental outcomes in preterm and full-term babies, as well as correlate pathogenetic periods, etiological factors, and clinical parameters to neuroradiology. The frequency was 0.66 per 1000 between the ages of 6 and 15. Postnatally acquired hemiplegia accounted for 11% of the total, with the majority of cases being postinfectious, iatrogenic, or posttraumatic. Prenatal (primarily circulatory brain lesions and maldevelopments) was the aetiology in 42 percent of term children with congenital hemiplegia (pre and perinatally derived), combined pre and perinatal in 9 percent, perinatal (cerebral haemorrhage, hypoxia) in 16 percent, and untraceable in 34 percent.
The overall handicap was classified as light in 40% of the cases, moderate in 44%, and severe in 16%. Postnatal cases had the highest rate of severe complete disability. In 109 congenital instances, Computerised Tomography (CT) revealed cortical/subcortical cavities in 20%, unilateral ventricular enlargement in 36%, and cortical/subcortical cavities in 26%. The remaining 18% of CT findings were categorised as "other." The associations between CT findings and aetiologies were inadequate and disappointing with the categories utilised so far. CT findings, on the other hand, were found to have a substantial association with the clinical severity of the impairment and its size. Normal CT was typically associated with mild disability and moderate unilateral ventricular enlargement, whereas cortical/subcortical cavities were usually associated with severe impairment, such as mental retardation and epilepsy. ABOUT THE STUDY Preterm children had a similar distribution, with 29 percent, 47 percent, 25 percent, and 6%, respectively. Preterm birth was found to be 24 percent of the time in congenital instances. Birth asphyxia was found to be a poor predictor of pathogen etic phase, but a series of postpartum problems pointed to perinatal brain injury. A clinical follow-up of 152 children found that 50% had mild motor dysfunction, 31% had moderate motor. Dysfunction and 19% had severe motor dysfunction. In 44 percent of the children, stereo gnostic sensibility was affected (astereognosia in 20 percent). Additional disabilities (mental retardation, epilepsy, impaired vision, hearing and speech, severe behavioural/perceptual issues) were found in 42 percent of the participants. Preterm children with congenital hemiplegia were more seriously affected than term children with congenital. Classification Cerebral palsy is classified based on how it affects people's movement, the body portion affected, and the severity of the effects [1].Ataxic cerebral palsy: Clumsiness, imprecision, and instability are all characteristics of ataxic movements. The movements are not smooth and may appear choppy or dis organized. When a person with ataxia tries to make voluntary motions like walking or picking up objects, they experience incoordination. Ataxia is characterized by a loss of muscle control in the arms and legs, which leads to a loss of balance and coordination.